Fig. 2

Photomicrograph of the testis of the experimental groups on the 28th day of the experiment, stained with H&E (X200, Scale bar = 50µ) (A) Control group showed normal testicular histology as the seminiferous tubule lined with a multilayer of spermatogenic cells (SC) and impacted with sperm (arrow) with interstitial Leydig cells (LC). (B) Camel milk group showed normal testicular histology as the seminiferous tubule lined with a multilayer of spermatogenic cells (SC) and impacted with sperm (SP) with interstitial Leydig cells (LC). (C) Silymarin group showed normal testicular histology as the seminiferous tubule lined with a multilayer of spermatogenic cells (SC) and impacted with sperm (SP) with interstitial Leydig cells (LC). (D) Aflatoxin group showed the arrest of spermatogenesis in some seminiferous tubules (arrows) with degenerative changes of spermatogenic cells and interstitial edema (arrowhead). (E) Aflatoxin and camel milk group showed normal testicular histology as the seminiferous tubule lined with a multilayer of spermatogenic cells (SC) and impacted with sperm (SP) with interstitial Leydig cells (LC). (F) Aflatoxin and silymarin group showed normal seminiferous tubule with mild hyperplasia of the interstitial Leydig cells (arrow) (n = 5/group)