Fig. 1

Photomicrograph of the testis of the experimental groups on the 21th day of the experiment, stained with H&E (X200, Scale bar = 50µ) (A) Control group showed normal testicular histology as the seminiferous tubule lined with a multilayer of spermatogenic cells (SC) and impacted with sperm (SP) with interstitial Leydig cells (LC). (B) Camel milk group showed normal testicular histology as the seminiferous tubule lined with a multilayer of spermatogenic cells (SC) and impacted with sperm (SP) with interstitial Leydig cells (LC). (C) Silymarin group showed normal testicular histology as the seminiferous tubule lined with a multilayer of spermatogenic cells (SC) and impacted with sperm (SP) with interstitial Leydig cells (LC). (D) Aflatoxin group showed degenerative changes of the lining spermatogenic cells (SC) in the form of vacuoles (arrows) and hyalinization (H). (E) Aflatoxin group showed hyperplasia of the interstitial leydig cells with vacuolization of the entire leydig cells (arrow). (F) Aflatoxin and camel milk group showed normal testicular histology as the seminiferous tubule lined with a multilayer of spermatogenic cells (SC) and impacted with sperm (SP) with interstitial Leydig cells (LC). (G) Aflatoxin and camel mild group showed mild vacuolation of the 1st layer of spermatogenic cells (arrows) with impacted seminiferous tubules with sperm (n = 5/group)