Fig. 2

Magnetic resonance imaging (MRI) performed 21 days after traumatic brain injury. (A) MRI shows a decrease in the area of the hyperintense lesion (white arrow), with a portion of this lesion becoming hypointense (white arrowhead). (B) T1-weighted MRI shows a hyperintense lesion with weak enhancement after intravenous administration of a paramagnetic contrast agent (yellow circle). (C) In diffusion-weighted imaging (DWI), the signal that the hyperintense lesion in Day 1 is decreasing and approaching isointense compared to in Day 1. (D) Apparent diffusion coefficient (ADC) mapping reveals a hypointense cerebellar lesion, and the intensity did not change (white arrow). (E) The hypointense lesion became smaller in the T2*-gradient-recalled echo imaging, and the location of the lesion was consisted the location of the with hypointense lesion in T2WI (white arrow). By T2WI and T2*, the signal of the lesion initiated intracellular hemosiderin