Fig. 3

Magnetic resonance imaging (MRI), transversal view of dog 3, follow up examinations. T2weighted (T2W) imaging (a, e, i), Fluid attenuation inversion recovery (FLAIR) (b, f, j), T1W post contrast (c, d, g, h, k, l) on the level of the pituitary gland (a-c, e-g, i-k) and the level of the septal nuclei (d, h, l). MRI performed at initial presentation (a-d) with only a subfalcine mass present (arrow). MRI immediately after presentation due to peracute onset of clinical signs attributable to a lesion in the right cerebrum (e-h). The previously reported mass is of decreased size (arrow) but otherwise unchanged. An additional intraaxial lesion attributable to an ischemic infarction of the right middle cerebral artery is visible. It is sharply delineated and involved the right cerebral hemisphere (asterisk). Marked homogeneous contrast enhancement of the outer layers of the cerebral cortex in the affected brain region is apparent (hash). Control MRI 13 months after initial presentation (I-L): the mass is reduced in size (arrow). In the area of the former presumed ischemic infarction, loss of parenchyma causes increased volume of the right lateral ventricle (section)