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Table 1 Clinical and diagnostic findings in Vizsla polymyopathy

From: Clinical features of idiopathic inflammatory polymyopathy in the Hungarian Vizsla

Clinical and Diagnostic Findings in VIP

Number of dogs from Group 1

% of total Group 1 dogs (24)

Number of dogs from Group 2

% of total Group 2 (53)

Total number of CASES with this clinical signs or had this diagnostic test out of total 77 dogs

% of total cases (77)

Dysphagia (pharyngeal phase of deglutition)

20

83%

49

92%

69

90%

Drinking and eating difficulties (Oral phase of deglutition)

21

87%

48

91%

69

90%

Sialorrhea

21

87%

46

87%

67

87%

Masticatory muscle atrophy

20

83%

45

85%

65

84%

Regurgitation

19

79%

42

79%

61

79%

Trismus

3

12%

13

25%

16

21%

Masticatory myalgia

1

4%

8

15%

9

12%

Aspiration pneumonia

4

17%

13

25%

17

22%

Toxoplasma gondii serumantibody titre (tested for and negative)

14

58%

11

21%

25

32%

Neospora caninum serum antibody titre (tested for and negative)

15

62%

10

19%

25

32%

Elevated serum creatine kinase (>190 IU/L)

18

75%

23

43%

41

53%

Serum creatine kinase >1000

11

46%

14

26%

25

32%

Anti- 2 M antibody titre (Tested for and negative)

12

50%

17

32%

29

38%

Anti-acetylcholine receptor antibody titre (Tested for and negative)

16

67%

16

30%

32

42%

Electromyography (Performed and findings suggestive of myopathy)

15

62%

5

9%

20

26%

Electromyography (Performed and findings normal)

1

4%

2

4%

3

4%

Modality by which diagnosis Megaoesophagus was made

Radiographs

8

33%

18

34%

26

34%

Barium

1

4%

9

17%

10

13%

Fluoroscopy

4

17%

8

15%

12

16%